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March 22, 2017
Study Evaluates Effect of Surgeon and Hospital Volume on Mortality After EVAR and Open AAA Repair
March 22, 2017—Sara L. Zettervall, MD, et al published a study of the effect of surgeon and hospital volume on mortality after open and endovascular aneurysm repair (EVAR) of abdominal aortic aneurysms (AAAs) in the Journal of Vascular Surgery (JVS; 2017;65:626–634).
The background of the study is that although it is known that higher hospital and surgeon volumes are independently associated with improved mortality after open repair of AAA in the era before EVAR, the effects of both surgeon and hospital volume on mortality after EVAR and open repair in the current era are less well defined.
As summarized in JVS, the investigators studied Medicare beneficiaries who underwent elective AAA repair from 2001 to 2008. Volume was measured by procedure type during the 1-year period preceding each procedure and was further categorized into quintiles of volume for surgeon and hospital.
Multilevel logistic regression models were used to evaluate the effect of surgeon volume, accounting for hospital volume, on mortality after adjusting for patient demographic and comorbid conditions, as well as the analogous effect of hospital volume adjusting for surgeon volume. The multilevel models included random effects for surgeon and hospital to account for the clustering of multiple patients within the same surgeon and within the same hospital.
The study was composed of 122,495 patients who underwent AAA repair (open: 45,451; EVAR: 77,044).
The investigators reported that after EVAR, perioperative mortality did not differ by surgeon volume (quintile 1 [0–6 EVARs]: 1.8% vs quintile 5 [28–151 EVARs]: 1.6%; P = .29), but decreased with greater hospital volume (quintile 1 [0–9 EVARs]: 1.9% vs quintile 5 [49–198 EVARs]: 1.4%; P < .01).
After open repair, perioperative mortality decreased with both higher surgeon volume (quintile 1 [0–3 open repairs]: 6.4% vs quintile 5 [14–62 open repairs]: 3.8%; P < .01) and hospital volume (quintile 1 [0–5 open repairs]: 6.3% vs quintile 5 [14–62 open repairs]: 3.8%; P < .01).
After adjustment for other predictors, the investigators found that surgeon volume was not associated with perioperative mortality after EVAR (odds ratio [OR], 0.9; 95% confidence interval [CI], 0.7–1.1); however, hospital volume was associated with higher perioperative mortality (quintile 1: OR, 1.5; 95% CI, 1.2–1.9; quintile 2: OR, 1.3; 95% CI, 1.02–1.6; and quintile 3: OR, 1.2; 95% CI, 1.01–1.5, compared with quintile 5).
After open repair, higher surgeon volume was also associated with lower mortality (quintile 1: OR, 1.5; 95% CI, 1.3–1.8; quintile 2: OR, 1.3; 95% CI, 1.1–1.6; and quintile 3: OR, 1.2; 95% CI, 1.1–1.4, compared with quintile 5). Risk of mortality also was higher for patients treated at lower-volume hospitals (quintile 1: OR, 1.3; 95% CI, 1.1–1.5; quintile 2: OR, 1.3; 95% CI, 1.1–1.5; and quintile 3: OR, 1.2; 95% CI, 1.1–1.4, compared with quintile 5).
The study concluded that after EVAR, hospital volume is minimally associated with perioperative mortality, with no such association for surgeon volume. After open AAA repair, surgeon and hospital volume are both strongly associated with mortality. These findings suggest that open surgery should be concentrated in hospitals and surgeons with high volume, advised the investigators in JVS.
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